Hyperthermia and supraspinal fatigue

Research output: Contribution to journalReview articlepeer-review

Abstract

The development of hyperthermia has a profound influence on neuromuscular function, with alterations occurring at both the central and the peripheral level. From a central perspective, heat stress can lead to a reduction in voluntary muscle activation (i.e. hyperthermia-induced central fatigue). Voluntary activation is typically quantified by normalizing the amplitude of the force/torque increment induced by percutaneous muscle (PMS), motor nerve (MNS) or transcranial magnetic stimulation (TMS) during a maximal voluntary isometric contraction (MVC), to the force/torque elicited by the same stimulus on the muscle at rest. When additional force/torque is produced via PMS or MNS during an MVC, it indicates incomplete activation of the motor pathway or that the stimulated axons are discharging at subtetanic rates.
Original languageEnglish
Pages (from-to)1323-1324
Number of pages2
JournalExperimental Physiology
Volume101
Issue number11
DOIs
Publication statusPublished - 2016
Externally publishedYes

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