The more the merrier: Faecalibacterium prausnitzii in Crohn's disease

Claire L. O'Brien, Gwen E. Allison, Paul Pavli

Research output: Contribution to journalEditorial

3 Citations (Scopus)

Abstract

Crohn's disease (CD) is thought to result from an inappropriate, ongoing immune response that occurs in genetically susceptible individuals as a result of a complex interaction between luminal bacteria, environmental factors, and the mucosal immune system. Three theories of microbial etiopathogenesis have been proposed:1 (i) a dysbiosis (an imbalance between potentially “beneficial” and potentially “harmful” bacteria); (ii) the presence of an unidentified pathogen; and (iii) excessive bacterial translocation.
Original languageEnglish
Pages (from-to)757-759
Number of pages3
JournalJournal of Gastroenterology and Hepatology (Australia)
Volume28
Issue number5
DOIs
Publication statusPublished - May 2013
Externally publishedYes

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